Actinic keratosis occurs mainly in light-skinned people. Frequency depends on exposure to sun, your age, and proximity to equator. Some people develop numerous skin lesions in a single area. There is a connection between beta-papillomavirus and actinic keratosis.
<p><strong>Clinical Trials and Research Studies on Actinic Keratosis</strong></p>
<p>New York (USA), June 21, 2013</p>
<p><strong>Actinic Keratosis: Clinical Trials and Research Studies</strong></p>
<p>Actinic keratosis occurs mainly in light-skinned people. Frequency of occurrence depends on exposure to sun, your age, and locational proximity to equator. It occurs in body parts exposed to sun’s rays like face, forearms, legs, and back. Further, cumulative ultraviolet ray exposure accentuates its occurrence. Frequency increases with each decade of your life as skin lesions spread and expand very slowly and gradually. DNA analysis of skin cells within actinic keratoses shows specific mutations in key genes due to ultraviolet rays.</p>
<p>Actinic keratosis starts as a rough patch, more felt than seen. It gradually develops into an elevated plaque spreading across as skin lesions measuring various centimeters in diameter. Over a long period spanning years, these lesions develop into invasive squamous cell carcinoma. Occurrence of actinic keratoses starts in your forties or fifties aggregating in your later life. Actinic keratosis is regarded as start of squamous cell carcinoma rather than as a precancerous lesion.</p>
<p>Some people develop numerous skin lesions in a single area. These collide and cause actinic keratosis over a much wider area. Some of them develop projections above skin surface similar to a horn. These are called cutaneous horns. There is a connection between beta-papillomavirus and actinic keratosis. However, risk is higher when combined with other factors like age, skin color, and damage due to sun’s ultra-violet rays.</p>
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